Özet:

Purpura Fulminans (PF) is a rapidly progressive, common skin necrosis. Especially on the legs there are sharp-limited large ecimotic plaques and on them are hemorrhagic bulbs1.2. A 72-year-old woman, who was ill, was sent to the emergency clinic with a fire and shock in her legs that began three days ago. The patient has been using antibiotics for 10 days due to a urinary tract infection. In the examination in the cardiovascular surgery clinic, in the distal of the right lower end and in the pretibial area of the left lower end, in the diameter of 5*10 cm, the necrotic place of the erodic and relaxed bulbs were observed (Figure 1 and 2). Platelet 60 thousand/ml, INR 3.2 and other tests were normal. The patient diagnosed with PF was also recommended symptomatic treatment for injury, requiring sepsis and consultation of infectious diseases for ICE.PF is one of the acute, severe cutanate syndrome, seen with common intravenous congestion (DIC). The main cause in the newborn period is the auto-dominant transitional protein C and protein S deficiency1.3. In childhood; red, streptocoxic faringitis, mild infections or severe infections such as meningoxemia, but in adulthood more grams (-) bacteria associated with sepsis after PF occur. The common thrombosis that holds the venous system in PF is concerned. There is no adhesion to the artery system. Thus, it can be easily taken. In the clinic there are sharply limited large ecimotic plaques and hemorrhagic bulbs on them. Lesions can be sensitive and net-released. Distal settled people can go to gangrene by moving rapidly. The general condition of the patients is disturbed and the fever is high. The blood table is initially associated with hypercoagulability and subsequently hypercoagulability that occurs as a result of overconsumption1–4. In the distinctive diagnosis; leukocytoclastic vasculitis, trombotic tromositopenic purpura and kumadine necrosis should be considered. PF diagnosis is placed with the presence of clinical and histopathological findings as well as peripheral blood findings associated with the DIC. PF should be treated by joint work of several medical branches in burning and/or intensive care units. The underlying reason is to be attempted to eliminate. If an infectious infection is affected, it should be treated and secondary infection prevention should be carried out. The fluid-electrolyte balance against the risk of shock should beined, the infusion of heparin should be started to inhibit the process of intravascular coating4 . In addition to these treatments, the debridation of the necrotic tissues and the duo-genic wound care should be carried out. Despite all of these treatments, the mortality of PF is high, and in living cases, end amputations and large skin injections may be required. As a result, early diagnosis is important due to the high mortality in PF, which is quite rare and requires rapid and aggressive treatments when diagnosed. We want to point out that the treatment of the underlying disease with early diagnosis is important to reduce the mortality of the patient.