Özet:

Recurren is a transversal myelite; a picture that can be formed with various ethologies. It has been that HBs Ag carrier may be the cause of transversal myelitis, which should be called corticosteroid-response recurren. A 32-year-old male patient, who was in the clinic with a close of pain, swelling and weakness in both lower limbs that lasted for 15 days. The patient was first discovered that there were four myelite attacks that began with myelite, where paraparesis, urinary incontinence and levels of sensational loss occurred, and seen with a similar picture. The laboratory examinations in that time were also compatible with myelite. In the neurological examination conducted in the patient’s final application; paraparesis, spastisity, DTRs more manifest in the left were hyperactive, bilateral Babinski (+), bilateral Achille clonus (+), the feeling defect in T8 was identified. In laboratory studies, hemogram and biochemical examinations were found normal, except for ESR:102 mm/h. In BOS examination; appearance clear, protein: 25 mg/dl, glucose (glc): 47 mg/dl found, there was no reproduction in culture, Brucella and Sy:(-), the cell was not traced, oligoclonal band: (-), Ig G index: 0.26. CRP is 21.8 mg/dl. Other hepatitis markers except HBs Ag (+; 44,69) were negative. VEP was normal. In the West USG, neurogenic urine and secondary hydronephrosis were observed. The urodinamic was compatible with the detrussor sfinkter disynergy. In the localization that meets the level of T11-12 in the spinalist of the medulla, the maximum is 1 cm. The hyperintensity area was detected. Seven days 1 gr/day with IV prednisolone treatment was observed in strength loss and improvement in sensory levels. Hepatitis B Ag is to be the cause of recurren myelitis with immunological mechanisms. We have seen this association in the light of literature.

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