Özet:

A 52-year-old female patient was referred to the clinic with complaints of bilateral buttock and leg pain for the last one year. In the initial interview, the patient complained of bilateral buttock pain radiating to both legs without improvement despite the use of nonsteroidal anti-inflammatory drugs and physical therapy. Her complaints started after a traumatic injury. Upon detailed questioning, she mentioned she had fallen on her buttocks severely after adjusting the treadmill to a high speed and altering the treadmill’s incline settings during her first use. The patient’s medical history was not notable except for a cholecystectomy three years earlier. On physical examination, there was a full active range of motion of all the lower limb joints and the lower back. Bilateral hip movements were painful with bilateral hip flexion and internal rotation. The straight leg raise test was positive on the left and right at 60 and 50 degrees, respectively. Her neurological and neurovascular examinations were normal. In addition, complete blood count, routine blood biochemistry, and acute phase reactants were within normal range. Bilateral piriformis syndrome was suspected after exclusion of the other pathologies using electrodiagnostic studies and lumbar magnetic resonance imaging. Her physical examination was repeated. She had tenderness to external palpation over greater sciatic notches on both sides. Flexion+adduction+internal rotation, Beatty, and Pace tests were positive bilaterally. Pelvis magnetic resonance imaging documented that both sciatic nerves were increased in intensity in the axial and coronal short-tau inversion recovery sequences (Figure 1a and b). After obtaining informed consent, the patient was administered an ultrasound-guided injection of 4 mL lidocaine 2% and 1 mL betamethasone dipropionate into both piriformis muscles using a 22G 88-mm spinal needle.(1) After the procedure, her pain decreased substantially (pre- and post-treatment visual analog scale scores for pain were 8 and 4, respectively). In the light of these findings, the patient was diagnosed with bilateral piriformis syndrome. Further treatments, including the spray and stretch technique, were incorporated two times as the pain was maintained at a visual analog scale level of 4/10.(2) At the six-week follow-up, she was completely asymptomatic. On physical examination, piriformis-specific tests were also negative for both sides. Making differential diagnosis of sciatica may be quite complex. Although piriformis syndrome is known as a cause of sciatica, bilateral piriformis syndrome is a rare phenomenon. Our report suggests that piriformis syndrome following a trauma may be causally related to the development of bilateral sciatica.

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