Abstract:

Inflammatory intestinal diseases (IBH) mainly include ulcerative colitis (UC) and Crohn's disease (CH). It is often a chronic-visible picture that occurs in genetically sensitive people, whose etiology is unknown, primarily holding the gastrointestinal (GI) system. Anemia is a common complication that is often neglected in HIV patients and pathogenesis (inflammation, iron, B12 and folic acid deficiency, myelosuppression) is multifactorial. In inflammatory intestinal patients, the etiology of chronic disease anemia (KHA) is the deterioration of erythropoesis at different levels. Chronic disease anemia occurs with the retention of iron (RES) by macrophages and dentritic cells in the reticuloendotelyal system, which occurs with the breakdown of erythrosites, and the failure to supply it to the pre-erythroid cells. All of these mechanisms lead to "functional iron deficiency", although there is iron in the body, it cannot be used for erythropoesis. It is that this is due to a chronic inflammation in the body and is caused by the negative effects of the drugs used in the treatment of this disease. A remarkable increase in these diseases has been observed in recent years, and despite many experimental studies, there is no agent that can provide a complete treatment in the clinical practice. The treatment of IBH is largely medical treatment and there is no medical nutritional treatment created at the proof level yet. In this collection article, in addition to anemia pathogenesis that occurs frequently in the UK and CH, it is intended to review the recommended nutritional therapy in the remission of the disease in the light of literary data.

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