Özet:

Traumatic asphyxia (Perthes syndrome) is a clinical syndrome that develops as a result of severe torasic compression, characterized by cervicophasial cyanosis, pethesia and subconjunctival hemorrhage triade. In the presented case, the objective is to recognize the clinical findings resulting from the developing asphyxia after torakoabdominal trauma and to share the experience of the patient’s anesthesia management. A 34-year-old patient with traumatic asphyxia after a traffic accident, cyanosis, swelling, swelling and subconjunctival hemorrhage in the headache, chest areas, was taken to surgery due to tibia fractures. Since cervicophageal changes can be a cause of difficult entubation, when video-olaringoscopy is used, it has been found that the peritoneal area and the hypofarynx are swelling, swelling. Although pathophysology is not exactly explained, increased intratorasic pressure is accused as a result of mediastinal compression formed by trauma. The findings are related to the severity, duration and vital organ damage of the press. Treatment includes support therapy along with the treatment of existing pathologies. As a result, in addition to proper monitoring in anesthesia applications because of the surgeons that may be needed in these cases, it is important to have the necessary equipment prepared by assessing the difficulty of entubation. SUMMARY Traumatic asphyxia (Perthes syndrome); Is a clinical syndrome characterized by cervicofacial cyanosis, petechiae, and subconjunctival hemorrhage triad, with severe thoracic compression. When present, it is aimed to identify the clinical findings that occur as a result of asphyxia after thoracoabdominal trauma and to share experiences about the anesthesia management of the patient. A 34-year-old patient with traumatic asphyxia following trauma, with cyanosis, edema, petitellar and subconjunctival hemorrhage in the head and neck, thoracic regions, was excluded because of tibia fracture. When videolaryngoscopy was used, the peritonsillary region and the hypopharynx petechiae, edema were seen as cervicofacial changes could be the cause of difficult intubation. Pathophysiology is not fully explained, but increased intrathoracic pressure resulting from trauma-induced mediastinal compression is implicated. Findings are related to the severity of the press, the duration and damage to vital organs. Treatment includes treatment of existing pathologies as well as supportive treatment. As a result, it is important that surgeons who are needed in these cases should be able to evaluate the difficulty of intubation and provide necessary equipment in addition to appropriate monitoring in anesthesia applications.

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