Özet:

Crohn’s disease (CH) and ulcerative colitis (OC) are the cause of inflamatory cervical diseases (IBH) where genetic and environmental factors play an unknown role in pathogenesis. Worldwide UN incidence has been as 6.3-24.3/100.000, CH incidence 5-20.2/100.000 (1). In Turkey, according to data from the IBH association in 2007, the prevalence of IBH is 36/100,000. In the past few years there have been increasing developments in the treatment strategies for inflamed amateur abdominal disease, but significant needs are still not met. Patients are looking for more secure, immunomodulating and alternative therapies to immunosuppressive therapies, and in this sense, the modulation of the cervical microbiot in IBH seems conceptually attractive and realistic (2). However, the unconvincible results in the studies and publications have not yet shown sufficient evidence on the subject, and therefore the studies remain insufficient to evaluate the reflection of the findings obtained in the clinical practice. In this article, the ethopathogenetic and therapeutic relationship between IBH and cervical microbiotism has been tried to be reviewed in the light of current information.

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